MET and gastric cancer: Typically, the intracellular tyrosine kinase catalytic domain induces receptor activation and auto‐phosphorylation at Tyr1234 and Tyr1235, which are known docking sites for downstream signal transduction molecules.5 Our finding suggested that exosomal MET secreted from H. pylori‐infected gastric cancer cells existed stably in the tyrosine‐phosphorylated form.