Furthermore, miR-122 inhibition was linked to the development of hemochromatosis by up-regulating the expression of human hemochromatosis protein (Hfe), hemojuvelin (Hjv), bone morphogenetic protein receptor type 1A (Bmpr1a) and the hepcidin antimicrobial peptide (Hamp) (Castoldi et al., 2011). The gene discussed is BMPR1A; the disease is hemochromatosis type 1.