Our results are supported by a previous study [1] that conducted a comparative study of CD138 and Ki-67 expression in different types of ameloblastomas, identifying that the solid type has the lower percentage of expression of CD138 (40.2% versus 49.7% found in UA), and they concluded that this might be one of the reasons why solid ameloblastomas (SA) tend to be more aggressive than its unicystic variant [21]. The gene discussed is MKI67; the disease is ameloblastoma.