Noh et al. showed that RKIP activity and levels in cervical and stomach cancer cell lines and tissues are inversely correlated with endogenous levels of NICD and that RKIP overexpression resulted in significant NICD reduction with subsequent inhibition of NICD-mediated expression of EMT markers, including vimentin, N-cadherin, and Snail. This evidence concerns the gene PEBP1 and gastric neoplasm.