CDH1 and cancer: Given the important role of E-cadherin downregulation and epithelial-mesenchymal transition (EMT) in phenotypic changes underlying the acquired enhanced motility and invasion of epithelium-derived cancer cells [15,16,17,18], we analyzed the expression levels of E-cadherin on HT29 cells after 48 h treatment with natural and yeast-derived recombinant avenanthramides.