CRP and non-alcoholic fatty liver: Recently, we showed in three different models of NAFL that B[a]P and ethanol coexposure induced the appearance of a steatohepatitis-like state characterized by hepatocyte demise and increased expression of several inflammation markers such as interleukin 1β (IL1β), IL6, tumor necrosis factor α (TNFα), and C-reactive protein (CRP) [14].