Consistent with our findings, ATAC-seq analyses of the previously deposited data, GEO: GSE8992811 uncovered a substantial enrichment of AP-1 during epidermal wound healing (24.55%) compared to homeostatic epidermal (15.31%) and HF stem cells (4.73%) (Supplementary Figure 9C). The gene discussed is JUND; the disease is hydrops fetalis.