RUNX1 and acute lymphoblastic leukemia: These lncRNAs were more B-ALL-subtype specific than protein-coding genes and of note, this study confirmed the study by Fernando et al. [57], showing upregulation of BALR-1 and LINC00958 and increased BALR-2 expression in patients with EVT6/RUNX1 and MLL rearrangements, respectively.