NFKB1 and cytomegalovirus infection: In addition to immediate-early expression of the NFκB agonists IE1 and UL144, IE2 inhibits host NFκB signaling at all points during HCMV infection through a still-controversial model: either by blocking NFκB subunit dimer interactions or preventing subunit interactions with specific NFκB target promoters, e.g., IL-6 [32,111].