Cui et al. have reported that lncRNA CCAT1 promotes glioma tumourigenesis by sponging miR‐181b, thereby leading to derepression of its endogenous targets FGFR3 and PDGFRα; these findings point to a potential therapeutic target in glioma.23, 24 A recent study verified that LINC00312 is associated with miR‐197‐3p and modulates the expression of a miR‐197‐3p target, p120, thereby providing powerful evidence that LINC00312 enhances gene expression at the post‐transcriptional level.20 Here, we revealed a potential binding site within LINC00312 and miR‐21 by bioinformatic prediction. This evidence concerns the gene FGFR3 and central nervous system cancer.