KIM‐1, which is endogenously expressed at a very low levels, is a well‐established urinary biomarker for acute kidney injury and is also known to be positively correlated with tubular and tubulointerstitial injury as well as with glomerular damage.26, 50, 51, 52 This is in a very good agreement with our findings, as we could also detect glomerular expression of KIM‐1 by immunofluorescence staining and in murine glomeruli of our podocyte dedifferentiation assay. The gene discussed is HAVCR1; the disease is acute kidney injury.