The authors reported Th17, Th9, Th2 and Th22 activation in both lesional and nonlesional skin and demonstrated significantly higher induction of Th17‐related cytokines (IL‐17A, IL‐19, CCL20, LL‐37 and peptidase inhibitor 3) in children with AD compared with adults with AD and control patients.28 We did not demonstrate any statistically significant difference in IL‐17A levels in the SC or plasma of patients with AD compared with healthy controls. This evidence concerns the gene CAMP and Alzheimer disease.