CCL5 and glioblastoma: For example, pUS28-induced migration in smooth muscle cells (SMCs) enhances CCL5-induced chemotaxis [61,64], while pUS28-induced prosurvival signals in glioblastoma multiforme (GBM) cells could favor a more malignant phenotype [70,72,73,74,76], suggesting pUS28-mediated changes in the host cell environment influence disease-specific phenotypes associated with HCMV.