Altmann et al. identified autosomal recessive homozygous or compound heterozygous mutations in TRDN, encoding triadin, associated with LQTS, and themselves proposed that “triadin knockout syndrome” or “TRDN-mediated autosomal-recessive LQTS” should be used rather than “LQT17,” because of the atypical phenotype that was observed (57). The gene discussed is TRDN; the disease is familial long QT syndrome.