In support of this, in addition to HuR and hnRNP-K, HIV-1 infection altered the binding of multiple additional protein partners of lincRNA-p21 (assessed by IP-MS, Figures S2I,J), further underpinning our hypothesis that HIV-1 alters the lincRNA-p21/hnRNP-K interaction to avert apoptosis. The gene discussed is ELAVL1; the disease is HIV-1 infection.