We observed that the two models exhibiting low MAP17expression, TP40 and TP60, were insensitive to carboplatin treatment, while those models with the highest MAP17 expression, TP57 and TP79, were sensitive to the drug, with tumour growth inhibition (TGI) percentages of 49.5% and 66.4%, respectively (Fig. 3e). Here, PDZK1IP1 is linked to neoplasm.