For example, the production of CXC-motif chemokine ligand 12 (CXCL12) results in chemotaxis of pro-tumorigenic CD11b+ myeloid-derived cells [49], whereas the upregulation of CXCL9, CXCL10, and CXCL16 can attract anti-tumor effector T cells [50–52]. The gene discussed is ITGAM; the disease is neoplasm.