In terms of its mechanistic involvement in cancers, CEP55 increases anchorage‐independent growth, migration, and invasion in vitro and promotes tumor formation in nude mice, possibly through VEGFA‐PI3K/AKT signaling (Chen et al, 2007; Inoda et al, 2009; Hwang et al, 2013). The gene discussed is CEP55; the disease is neoplasm.