It can also act as a ceRNA for miR-195 and reverse miR-195-mediated EGFR inhibition and further promote cell proliferation by activating the PI3K/AKT and JAK/STAT pathways, indicating a role of MALAT1-miR-195-EGFR axis in HCC [50] (Table 1). This evidence concerns the gene AKT1 and hepatocellular carcinoma.