The study found that TET3 can block transforming growth factor β1 (TGF-β1) by demethylating the miR-30d precursor gene promoter.[37] Studies also showed that miR-30d functioned as a suppressor of ovarian cancer progression by decreasing Snail expression and thus blocking TGF-β1-induced EMT process.[38] The pooled studies can provide a reference for studying the mechanism of ovarian cancer and targeted therapy. The gene discussed is TET3; the disease is ovarian carcinoma.