FGF23 and hyperphosphatemia: Currently, the identification of the triad of hyperphosphatemia, αKlotho deficiency, and elevated FGF23 levels as being central in the pathophysiology of uremic cardiomyopathy, unravels uremic cardiomyopathy as a new piece in the CKD-MBD puzzle, paving the way for more specific therapeutic possibilities for these patients.