CDKN2B-AS1 and neoplasm: ANRIL overexpression promotes proliferation, migration, invasion, and epithelial-mesenchymal transformation but inhibits cell apoptosis; ANRIL loss-of-function represses tumor size and growth rate, cell proliferation, migration, invasion, metastasis, and enhances apoptosis and senescence (22, 38, 55, 56, 58, 77, 80, 81, 84, 89–92).