Various receptor tyrosine kinases such as Epidermal Growth Factor Receptor (EGFR), HGFR/c-Met along with their downstream signalling molecules, such as GRB2, mitogen-activated protein kinase (MAPK), and Phosphoinositide 3-kinase (PI3K), have been found to be over-expressed in lung cancer [17]. Here, EGFR is linked to lung carcinoma.