The largest molecular biology research on IBC mainly focused on the transcriptome and demonstrated the presence of molecular subtypes similar to those of non-IBC tumors, although with over-representation of human epidermal growth factor receptor 2 (HER2)-enriched tumors and a low prevalence of Luminal A tumors, and suggested the deregulation of the expression of few genes in IBC compared with non-IBC, in particular those involved in cell motility, invasion, inflammatory pathways, and transforming growth factor (TGF)Beta signaling [6–8]. Here, TGFB1 is linked to inflammatory breast carcinoma.