Nevertheless, 35 days after modeling TBI, we found that the TBI model group displayed a significantly lower NeuN+/BrdU+/c-Fos+ cell count than the Sham group, suggesting that fewer immature neurons survived to become functional mature neurons in the post-TBI microenvironment, which may consequently contribute to memory impairment. The gene discussed is RBFOX3; the disease is memory impairment.