Using in silico tools, we identified significant overlaps between EEF1A1, UBC,UBB, CALR, CXCR4, JUND, FOS, PIM2, JUN, GAPDH, and HSP90B1, which were previously reported to be upregulated in MM according to the Munıshı_ MM data set (Figure 4). This evidence concerns the gene JUN and Miyoshi myopathy.