SLC26A4 and nodular sclerosis classical Hodgkin lymphoma: These findings corroborate with many studies with NSHL patients [19–21] that report up to 61% of cases presenting heterozygous mutations in the SLC26A4 gene and suggest that these mutations might contribute to phenotype due to the presence of other mutations not detected (in genes or in regulatory regions not investigated), which together affect pendrin expression in the inner ear.