To contribute to a better understanding of HML-2 transcription and HML-2 proteins potentially expressed in the ALS context, we identified transcribed HML-2 loci by employing previously established Sanger sequencing-based strategies [17, 29, 30] that target three different regions of the HML-2 proviral genome and using primer sets compensating for nt differences between HML-2 loci within respective primer binding regions. The gene discussed is CLEC10A; the disease is amyotrophic lateral sclerosis.