They also reported that FGF12 single-knockout (KO) mice showed no phenotype, while FGF12/FGF14 double-KO mice exhibited severe ataxia and decreased excitability of cerebellar granule neurons, thereby concluding that FGF12 played an important role together with FGF14 in neuronal action potentials. This evidence concerns the gene FGF14 and cerebellar ataxia.