PPARGC1A and myeloid sarcoma: Future work will include analyses of expression of additional Nrf2 and PGC1α target genes, examination of vascular oxidative stress associated with MS, exploration of whether reversal of the identified deficits in antioxidant response of MS‐MSC improves the neuroprotective potential of MS‐MSC, and further analysis of bone marrow microenvironment and function in MS.