They also identified a Let‐7f binding site in Cyp19a1, indicating that the aromatase gene is a direct target of Let‐7f.43 We demonstrated similar results in endometrial stromal cells from endometriosis patients and in Ishikawa cells.17 In our study, significantly increased Let‐7b and Let‐7f expression levels were determined after aromatase inhibitor treatment. This evidence concerns the gene CYP19A1 and endometriosis.