Because C. elegans adult somatic cells have the unique condition of little caspase or CEP-1, and ATM usually induces apoptosis via p53 in mammalian cells [1, 36], and abnormality of ATM induces ataxia-telangiectasia in humans (3), atm-1(tm5027) did not have obvious abnormalities in behavior. This evidence concerns the gene TP53 and ataxia telangiectasia.