EGFR and neoplasm: Xu et al. [16] reported that tumours with double common EGFR mutations (19Del + L858R, n = 18) exhibited similar antitumour responses to small-molecule TKIs as tumours with single common mutations, and the median PFS and ORR rates were 9.53 months and 71.4% (10/14), respectively, which is consistent with our results (10.1 months and 60%, respectively) and those of Hata Akito (16.5 months and 86%, respectively) [11].