Infection with the ΔgliP mutant significantly stimulated the expression of CXCL2, CCL3, CCL4, CCL7, TNF-α, and IL-6, whereas infection with the Δaspf1 ΔgliP double mutant significantly decreased the expression of CXCL2, CCL7, TNF-α, and IL-6. The gene discussed is TNF; the disease is infection.