Investigation of the monogenic form of FH includes testing for pathogenic variants in the genes for LDLR, ApoB, PCSK9 (related with the autosomal dominant type), and LDLRAP1 (autosomal recessive form) (Harada-Shiba et al., 2012a; Ito and Watts, 2015). Here, APOB is linked to familial hyperaldosteronism.