Moreover, GWAS offered a broader insight into the epidemiology of T2D by identifying potential genetic links between lipid dysregulation and glycaemia (FADS1, HNF1A, and GCKR), circadian rhythmicity and metabolic alterations (MTNR1B and CRY2), and between low birth weight and subsequent T2D risk (ADCY5) [10,16]. This evidence concerns the gene FADS1 and type 2 diabetes mellitus.