For examples, studies have shown that β-catenin plays a decisive role in nasopharyngeal carcinoma stem cells through the regulation of the EGFR / AKT gene [12], and overexpression of β-catenin has been considered as a biomarker of epithelial–mesenchymal transition (EMT) in prostate stem cells [13]. This evidence concerns the gene AKT1 and nasopharyngeal carcinoma.