GJB1 and peripheral neuropathy: Nonetheless, Cx32-null mice develop a late-onset peripheral neuropathy with demyelination features similar to those found in humans with Cx32 mutations (Nelles et al., 1996; Anzini et al., 1997; Scherer et al., 1998), indicating that other functions performed by Cx32 in SCs could be involved in the pathogenesis of the disease.