METTL14 and hepatocellular carcinoma: The clinical data also revealed that patients with decreased METTL14 showed worse recurrence‐free survival and overall survival.50 METTL14 deficiency enhanced the metastatic capacity of HCC cells, and conversely, overexpression of METTL14 suppressed cell migration and invasion.50 Further experiments demonstrated that pri‐miR‐126 was a direct target of METTL14 with m6A modification and m6A‐modified pri‐miR‐126 bound to DGCR8 to induce the expression of mature miR‐126 consequently.