Maintains the stability of the EBV genomes in the infected cells (37)Reduces p53 levels and promotes cell survival (38)Suppresses TGF-β1 signaling and promotes oncogenesis (39)Expressed in memory B cells undergoing division (40)Inactivation reduces the copy number of the episomes in EBV-infected B lymphoma cells in vitro and inhibits growth (41)Overexpression increases the nuclear levels of metastatic proteins like mapsin, Nm23-H1, and stathmin1 in NPC (42). This evidence concerns the gene NME1 and nasopharyngeal carcinoma.