Knockout of c‐met within mice models could give rise to embryonic death, and c‐met was implied to advance embryogenesis through curbing epithelial‐mesenchymal transition (EMT) process of muscle‐derived stem cells, as well as development of neuronal precursors, hepatic tissues and placenta tissues.31 Nonetheless, hardly any investigations have pointed out whether HOAIR, miR‐613 and c‐met would cooperate to alter the EMT process of retinoblastoma. This evidence concerns the gene MET and retinoblastoma.