JAK/STAT signaling has been heavily implicated in tumor progression [5,6,7], as have other receptor tyrosine kinases (RTKs), such as those associated with platelet-derived growth factor (PGDF) [8], vascular endothelial growth factor (VEGF) [9,10], and epidermal growth factor (EGF) [11]. This evidence concerns the gene EGF and neoplasm.