HNF1A-MODY is characterized by progressive glucose dysregulation, clinically presented at first with postprandial, later fasting hyperglycaemia, usually during adolescence or early adulthood (1, 8); thus, a large glucose increment of > 5 mmol/L can be observed on oGTT (12) in the early stages of the disease, whereas fasting blood glucose levels remain normal (14). The gene discussed is HNF1A; the disease is Hyperglycemia.