FCGR2A and HIV-1 infection: The hypothesis that CD32 cell surface expression would allow for the selective recognition of an HIV-1 reservoir contradicts the paradigm of the “undistinguishable phenotype of latently infected cells”; however, the data could be interpreted as indicating that latently infected cells would no longer be resting as HIV-1 infection leads to expression of an immune modulator (CD32) and likely 103 other genes, as suggested by Descours et al.12.