PVALB and schizophrenia: Mice hypomorphic in the N-methyl-d-aspartate (NMDA) R1 subunit (NR1) exhibited decreased PPI and social affiliation64, but the induction of schizophrenia-like symptoms by partial ablation of NR1 from GABAergic neurons, a majority of which were parvalbumin-positive, pointed to the mediation of schizophrenia-like phenotypes by NMDA-receptors on GABAergic neurons65, in which case the schizophrenia-like phenotypes could stem from disturbance of either the NMDA-receptors or GABAA-transmission or both.