A zebrafish model of CLN3 disease or Batten's Disease was evaluated for seizure susceptibility, which revealed the first sign of motor abnormality at 36 h post-fertilization (hpf) whereby the CLN3 ATG morphants displayed higher movement activity and a higher number of tail flicks, both of which are indicative of seizure activity (Packer et al., 2016). Here, CLN3 is linked to juvenile neuronal ceroid lipofuscinosis.