Li et al reported that the expression of HOXA11‐AS in breast cancer tissue was higher than that in tissue adjacent to cancer.27 Interfering in HOXA11‐AS could stimulate the apoptosis of breast tumor cell as well as inhibit the invasion and migration capacity of tumor cell through affecting EMT‐related molecular markers expressions such as E‐cadherin, N‐cadherin, Vimentin.27 This evidence concerns the gene HOXA11 and neoplasm.