A previous study demonstrated that FBP1 was a negative regulator of EMT in GC.18 Loss of FBP1 by Snail‐mediated repression provided metabolic advantages in basal‐like breast cancer.25 The ectopic expression of aldolase B was associated with poor prognosis and promotes tumour progression by EMT in colorectal adenocarcinoma.17 Some oncogene and tumour suppressors had a crosstalk between EMT and glycolysis in cancer progression and metastases, such as HIF‐1α,23 c‐myc,26 FOXM1,27 Gas1,28 and so on. This evidence concerns the gene FOXM1 and colorectal adenocarcinoma.