This is interesting because GWA studies associated BACH2 with IgG galactosylation [49] as well as with various immune and inflammatory diseases including IBD [4, 5, 65–67] in which IgG acquires pro-inflammatory properties through decrease in galactosylation, sialylation, and fucosylation [43, 46]. The gene discussed is BACH2; the disease is inflammatory bowel disease.