As the level of degranulation in response to primary MM cells was very low and this could have blunted analysis of inhibitory effects by NKG2A, we blocked the HLA-E–NKG2A interaction with an NKG2A blocking antibody (Figure 3C) or with an anti HLA-E antibody (Figure S6 in Supplementary Material). The gene discussed is HLA-E; the disease is Miyoshi myopathy.